Background Diesel exhaust particles (DEPs) are the main component of traffic-related air
pollution and have been implicated in the pathogenesis and exacerbation of asthma.
However, the mechanism by which DEP exposure aggravates asthma symptoms remains
unclear. Objective This study aimed to identify a key cellular player of air pollutant-induced
asthma exacerbation and development. Methods We examined the distribution of innate
immune cells in the murine models of asthma induced by house dust mite and DEP …